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In the Fontan circulation, the blood flows passively through the human body into the lungs due to the absence of the left heart. The etiology of PLE is not known. The prevalence of PLE appears.
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(2017, May 31). All heart patients have some liver disease after fontan surgery: Researchers report universal liver fibrosis in survivors of operation for a severe heart defect. ScienceDaily.
and recurrence of PLE even after OHT has been reported. Early diagnosis of other life-threatening Fontan complications, such as plastic bronchitis, is crucial to allow for effective medical therapy,
We describe the case of an adult who developed protein-losing enteropathy 21 years after having undergone a Fontan procedure. Investigations revealed iron deficiency anemia which was treated with.
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Catch-up growth in weight occurs after the Fontan and Mustard operations. In the Mustard population, height also normalizes, whereas in patients with univentricular circulation, height remains.
Cirrhosis with or without abnormal hepatic nodules was seen in 29 of 53 patients (55%) at 18.4 ± 5.6 years after the Fontan procedure. Adverse hepatic-related outcome occurred in 22% of the entire.
BACKGROUND Patients with protein-losing enteropathy (PLE) following the Fontan operation have a reported 50% mortality at 5 years after diagnosis. OBJECTIVES The aim of this study was to review.
Micropulsatility limited the effects of pulsatility loss. shown to be correlated with increased pulmonary vascular resistance and failing circulation after Fontan operations . We also found.
Determinant risk factors for developing protein losing enteropathy (PLE), including haemodynamics, remain unclear in patients after the Fontan operation. Our purpose was to characterize the serial PLE.
It is well-recognised that improved strength is associated with higher levels of performance in health and disease [21,25] but our findings suggest that, in the Fontan group, gains in strength are.
. fenestration to create an interatrial communication has been used to treat patients with protein losing enteropathy (PLE) after Fontan operation. No systematic data have been reported assessing.
In a case report, sildenafil has been associated with the resolution of protein-losing enteropathy after the Fontan procedure.  Sildenafil is often used in combination with extracorporeal membrane.
Although the increased CVP in Fontan patients has been associated with protein-losing enteropathy, hepatic dysfunction, and arrhythmias    , its effect on the kidneys has not been defined.
1, 2) PLE has been reported in 5 to 13% of patients after Fontan opera- tion, which subsequently to Glenn operation has been applied for the treatment of patients with congenital heart disease such as.
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Abnormal height and adiposity are observed after the Fontan operation. These abnormalities may be. which may have significant long-term implications in this at-risk population. Our study.
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Fifteen months following the Fontan operation, he developed a PLE and Fontan failure requiring OHT. Four years after OHT, he developed a severe tricuspid regurgitation and a PLE. His PLE improved.
2 In this study, the median time interval between Fontan-type surgery and the occurrence of PLE was 2.7 years (range: 1 month to 16.4 years). Our patient developed PLE 14 years after the Fontan.
Background Little is known about the enteric protein loss in patients after a modified Fontan operation before the appearance of overt symptoms or signs of protein-losing enteropathy (PLE). The.
The current study was undertaken to assess the frequency of excessive enteric protein loss and protein-losing enteropathy in the relatively early period after the Fontan operation. Protein excretion.